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Decoupling AMPK from fatty acid synthesis allows maintenance of fitness late in life
['Hanane Hadj-Moussa', 'Megan Ulusan', 'Dorottya Horkai', 'Mohammed Kamran Afzal Mirza', 'Jonathan Houseley', 'Hadj-Moussa H', 'Ulusan M', 'Horkai D', 'Houseley J', 'Allen C']
eLife: latest articles
Figure 2 with 1 supplement with 1 supplement see all Download asset Open asset Combining AMPK activity and fatty acid synthesis to avoid senescence.
(F) Size of colonies formed in 24 hr on a YPD plate by log phase and age-matched wild type and A2A.
While young A2A cells formed slightly smaller colonies than did wild-type cells, the colonies formed by aged A2A cells were much larger than those formed by wild-type cells of equivalent replicative age (Figure 2F).
Indeed, aged A2A cells formed colonies of similar size to young A2A cells, showing equivalent fitness, whereas most aged wild-type cells divided only once or not at all across the 24-hr assay period.
This demonstrates that A2A cells maintain excellent fitness late in life, whereas the fitness of aged wild-type cells is very low.